How do sulfa antibiotics sulfonamides kill bacteria in plants?

Caesar Fritsch asked a question: How do sulfa antibiotics sulfonamides kill bacteria in plants?
Asked By: Caesar Fritsch
Date created: Mon, Feb 8, 2021 12:49 PM

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Those who are looking for an answer to the question «How do sulfa antibiotics sulfonamides kill bacteria in plants?» often ask the following questions:

❓ How do sulfa antibiotics sulfonamides kill bacteria?

  • The sulfa drugs such as sulfonamides inhibit a critical enzyme--dihydropteroate synthase--in this process. Once the process is stopped, the bacteria can no longer grow. Another kind of antibiotic--tetracycline--also inhibits bacterial growth by stopping protein synthesis.

❓ How do sulfa antibiotics sulfonamides kill bacteria naturally?

Similarly, it is asked, how do sulfonamides kill bacteria? Sulfa drugs are bacteriostatic; i.e., they inhibit the growth and multiplication of bacteria but do not kill them. They act by interfering with the synthesis of folic acid (folate), a member of the vitamin B complex present in all living cells. Secondly, how do sulfonamides work?

❓ How do sulfa antibiotics sulfonamides kill bacteria in food?

Sulfonamides are bacteriostatic drugs, which inhibit the growth and reproduction of bacteria by interfering with the metabolism of bacterial folate. Unlike human and mammalian cells, bacteria that are sensitive to sulfa drugs cannot directly use folic acid in the surrounding environment.

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Mode of Action of Sulfonamides: The mode of action of sulfonamides is the inhibition of tetrahydrofolic acid (THFA) synthesis. Many bacteria require p-aminobenzoic acid (PABA) as a precursor to their synthesis of the essential coenzyme tetrahydrofolic acid (THFA). PABA is a structural part of the THFA acid molecule.

Sulfonamides are the sulfa drugs and serve as a competitive inhibitors of p -aminobenzoate (PABA). PABA is one of the substances involved in the synthesis of folic acid. sulfonamides compete with PABA for the enzyme of folic acid synthesis and thereby, block the pathways. In the absence of folic acid, bacterial growth is inhibited.

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